Your privacy, your choice

We use essential cookies to make sure the site can function. We also use optional cookies for advertising, personalisation of content, usage analysis, and social media.

By accepting optional cookies, you consent to the processing of your personal data - including transfers to third parties. Some third parties are outside of the European Economic Area, with varying standards of data protection.

See our privacy policy for more information on the use of your personal data.

for further information and to change your choices.

Skip to main content
Fig. 6 | BMC Immunology

Fig. 6

From: PU.1-CD23 signaling mediates pulmonary innate immunity against Aspergillus fumigatus infection by driving inflammatory response

Fig. 6

The expressions of PU.1, CD23, p-ERK, CCL20 and IL-8 in Ad-PU.1/Ad-CD23 mouse lung tissues under AF infecton and NS treatment. A Immunochemistry results showed the protein expressions of PU.1 and CD23 in mouse lung tissues injected with Ad-PU.1 and Ad-CD23 adenovirus with AF infection compared to the NS conditions. B, C The quantitative analysis of PU.1 and CD23 expressions in A. NS conditions and empty vector served as controls. D The western blotting analysis showed the protein levels of PU.1, CD23, p-ERK, CCL20 and IL-8 in mouse lung tissues injected with Ad-PU.1 adenovirus with AF infection compared to the NS treatment. The samples derived from the same experiment and that gels/blots were processed in parallel. EI The quantification of protein levels in D. All data are presented as the mean ± SD, N ≥ 3, *P < 0.05, **P < 0.01. Bars = 200 μm

Back to article page